Actively Recruiting
Comparative Evaluation of Melatonin Versus MTA on Vital Pulp Therapy in Young Permanent First Molars: An in Vivo Study
Led by Suez Canal University · Updated on 2024-08-02
45
Participants Needed
1
Research Sites
71 weeks
Total Duration
On this page
AI-Summary
What this Trial Is About
The process of dental caries is dynamic and can be either reversible or irreversible depending on the balance between protective and pathologic factors in the oral cavity. Untreated dental caries causes pulpal injury, inflammation, and necrosis. Melatonin plays an essential role in the regulation of bone growth. The actions that melatonin exerts on odontoblasts may be similar to its action on osteoblasts.
CONDITIONS
Official Title
Comparative Evaluation of Melatonin Versus MTA on Vital Pulp Therapy in Young Permanent First Molars: An in Vivo Study
Who Can Participate
Eligibility Criteria
You may qualify if you...
- Children aged 6 to 8 years who are apparently healthy and medically free
- Both boys and girls are included
- Children must have mandibular permanent first molars that can be restored
- No clinical signs of pulp degeneration, including no spontaneous or night pain, no pain on tapping, no tooth mobility
- No abscess, purulent sinus, or soft tissue swelling
- Bleeding from pulp should stop within 5 minutes after pulp amputation
You will not qualify if you...
- Internal root resorption
- External root resorption
- Bone destruction around or between roots seen on X-rays
- Deep carious lesion close to the pulp seen on X-rays
AI-Screening
AI-Powered Screening
Complete this quick 3-step screening to check your eligibility
Trial Site Locations
Total: 1 location
1
Faculty of Medicine, Suez canal university
Ismailia, Egypt, 41522
Actively Recruiting
Research Team
M
Mennatallah Hesham El shaabany, doctorate student
CONTACT
A
Asmaa Ali Abo El-Soud, Associate professor
CONTACT
How is the study designed?
Study Type
INTERVENTIONAL
Masking
SINGLE
Allocation
RANDOMIZED
Model
PARALLEL
Primary Purpose
TREATMENT
Number of Arms
3
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