Actively Recruiting

Age: 18Years +
All Genders
Healthy Volunteers
NCT07291908

Inflammation Severity and miRNA-126 in Trauma

Led by Melike Cengiz · Updated on 2025-12-18

130

Participants Needed

1

Research Sites

81 weeks

Total Duration

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AI-Summary

What this Trial Is About

Trauma triggers a complex immune response intended to eliminate danger signals and restore physiological balance. Early post-traumatic inflammation is primarily initiated by damage-associated molecular patterns (DAMPs) and pathogen-associated molecular patterns (PAMPs). In patients with severe trauma, dysregulated inflammation increases susceptibility to infection, systemic inflammatory response syndrome (SIRS), multiple organ dysfunction syndrome (MODS), and mortality. The lungs are particularly vulnerable, and excessive inflammatory activation may lead to acute lung injury (ALI) or acute respiratory distress syndrome (ARDS), conditions characterized by increased vascular permeability, alveolar epithelial injury, surfactant dysfunction, and impaired gas exchange. Pro-inflammatory cytokines, activated neutrophils, reactive oxygen species, and proteases contribute to endothelial and epithelial barrier disruption. Recent evidence also suggests that several microRNAs, including miR-126, may play a regulatory role in pulmonary barrier integrity through modulation of tight-junction proteins and PI3K/AKT-related pathways. Although many components of the trauma-related inflammatory response have been described, the relationship between systemic inflammatory severity and impairment of pulmonary gas exchange remains insufficiently defined in clinical settings. This study aims to investigate the correlation between inflammatory severity markers (C-reactive protein, procalcitonin, IL-6, reactive oxygen derivatives, neutrophil-to-lymphocyte ratio, lactate), imaging findings (flow-mediated dilation by ultrasound), clinical parameters (blood pressure, heart rate, urine output, vasoactive medication requirements), pulmonary gas-exchange measurements (arterial blood gases, PaO₂/FiO₂ ratio), and circulating miRNA-126 levels in trauma patients. The findings may help identify biomarkers that better reflect inflammatory burden and the risk of lung dysfunction following trauma.

CONDITIONS

Official Title

Inflammation Severity and miRNA-126 in Trauma

Who Can Participate

Age: 18Years +
All Genders
Healthy Volunteers

Eligibility Criteria

Eligible

You may qualify if you...

  • Adults aged 18 years or older
  • Patients monitored and treated for trauma in the anesthesia intensive care units of Akdeniz University Faculty of Medicine
Not Eligible

You will not qualify if you...

  • Patients younger than 18 years
  • Patients with concomitant thoracic trauma
  • Presence of active infection prior to trauma
  • Patients not admitted to the ICU within the first 24 hours after trauma
  • Patients who remain in the ICU for less than 72 hours following trauma
  • Current use of steroids, chemotherapy, or antibiotic therapy prior to ICU admission
  • Patients with immunodeficiency
  • Patients who are in shock prior to or during ICU admission

AI-Screening

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Trial Site Locations

Total: 1 location

1

Akdeniz University Hospital

Antalya, Ağrı, Turkey (Türkiye), 04200

Actively Recruiting

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Research Team

M

Melike Cengiz, Prof Dr

CONTACT

C

Canberk Kurban

CONTACT

How is the study designed?

Study Type

OBSERVATIONAL

Masking

N/A

Allocation

N/A

Model

N/A

Primary Purpose

N/A

Number of Arms

3

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